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Overall, our histological findings are consistent with publicly available transcriptomic data of the mouse large intestines obtained from the Tabula Muris 71 (Fig

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High blood glucose levels or glucose fluctuations can lead to vascular endothelial dysfunction and atherosclerosis through mechanisms such as increased oxidative stress, the formation of advanced glycation end products (AGEs), and the activation of protein kinase C (PKC) (Brownlee, 2001