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A total of 1961 participants were randomized in the trial, with 1,306 assigned to the semaglutide group and 655 to the placebo group

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Identification of the mechanisms linking GLP-1R signaling to control of BP and the biology of renal artery and kidney protein expression extends our understanding of GLP-1 action and highlights the importance of the VSMC as a key GLP-1R + cell type contributing to the cardiorenal actions of GLP-1 medicines such as semaglutide